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Properties of local synapses were analyzed in lobus parolfactorius (LPO; avian homologue of caudate-nucleus) of quail chicks by using slice preparations in vitro. Field-potential extracellular and whole-cell intracellular recordings revealed excitatory synaptic inputs converging from dorsal and ventral regions within LPO. With exogenous dopamine (100 microM) in the perfusate, synchronized conditioning stimulation induced biased changes in the dorsal and the ventral inputs; potentiation in the dorsal input and depression in the ventral input in average. On the other hand, de-synchronized conditioning failed to induce such biased changes, although the differences were not statistically significant. SCH-23390 (3 microM) blocked the dorsal potentiation, while AP-5 (100 microM) tended to block both of these changes. The plastic nature may underlie the memory formation in appetitive/aversive learning tasks.

Titel:	D1-receptor dependent synaptic potentiation in the basal ganglia of quail chicks.
Autor/in / Beteiligte Person:	Matsushima, T ; Izawa, EI ; Yanagihara, S
Link:	Full Text Finder (Volltext)
Zeitschrift:	Neuroreport, Jg. 12 (2001-09-17), Heft 13, S. 2831-7
Veröffentlichung:	London, England : Lippincott Williams & Wilkins ; <i>Original Publication</i>: Oxford, UK : Rapid Communications of Oxford Ltd., [1990-, 2001
Medientyp:	academicJournal
ISSN:	0959-4965 (print)
DOI:	10.1097/00001756-200109170-00016
Schlagwort:	Action Potentials drug effects Action Potentials physiology Aging physiology Animals Animals, Newborn anatomy & histology Animals, Newborn metabolism Avoidance Learning drug effects Basal Ganglia drug effects Basal Ganglia metabolism Benzazepines pharmacology Conditioning, Psychological Dopamine metabolism Dopamine pharmacology Dopamine Antagonists pharmacology Electric Stimulation Excitatory Amino Acid Antagonists pharmacology Excitatory Postsynaptic Potentials drug effects Excitatory Postsynaptic Potentials physiology Long-Term Potentiation drug effects Memory drug effects Neurons drug effects Neurons metabolism Organ Culture Techniques Quail anatomy & histology Quail metabolism Reaction Time drug effects Reaction Time physiology Receptors, Dopamine D1 drug effects Receptors, Glutamate drug effects Receptors, Glutamate metabolism Synapses drug effects Synaptic Transmission drug effects Synaptic Transmission physiology Avoidance Learning physiology Basal Ganglia growth & development Long-Term Potentiation physiology Memory physiology Quail growth & development Receptors, Dopamine D1 metabolism Synapses metabolism
Sonstiges:	Nachgewiesen in: MEDLINE Sprachen: English Publication Type: Journal Article; Research Support, Non-U.S. Gov't Language: English [Neuroreport] 2001 Sep 17; Vol. 12 (13), pp. 2831-7. MeSH Terms: Avoidance Learning / physiology ; Basal Ganglia / growth & development ; Long-Term Potentiation / physiology ; Memory / physiology ; Quail / growth & development ; Receptors, Dopamine D1 / metabolism ; Synapses / *metabolism ; Action Potentials / drug effects ; Action Potentials / physiology ; Aging / physiology ; Animals ; Animals, Newborn / anatomy & histology ; Animals, Newborn / metabolism ; Avoidance Learning / drug effects ; Basal Ganglia / drug effects ; Basal Ganglia / metabolism ; Benzazepines / pharmacology ; Conditioning, Psychological ; Dopamine / metabolism ; Dopamine / pharmacology ; Dopamine Antagonists / pharmacology ; Electric Stimulation ; Excitatory Amino Acid Antagonists / pharmacology ; Excitatory Postsynaptic Potentials / drug effects ; Excitatory Postsynaptic Potentials / physiology ; Long-Term Potentiation / drug effects ; Memory / drug effects ; Neurons / drug effects ; Neurons / metabolism ; Organ Culture Techniques ; Quail / anatomy & histology ; Quail / metabolism ; Reaction Time / drug effects ; Reaction Time / physiology ; Receptors, Dopamine D1 / drug effects ; Receptors, Glutamate / drug effects ; Receptors, Glutamate / metabolism ; Synapses / drug effects ; Synaptic Transmission / drug effects ; Synaptic Transmission / physiology Substance Nomenclature: 0 (Benzazepines) ; 0 (Dopamine Antagonists) ; 0 (Excitatory Amino Acid Antagonists) ; 0 (Receptors, Dopamine D1) ; 0 (Receptors, Glutamate) ; VTD58H1Z2X (Dopamine) Entry Date(s): Date Created: 20011006 Date Completed: 20011204 Latest Revision: 20191210 Update Code: 20240513

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D1-receptor dependent synaptic potentiation in the basal ganglia of quail chicks.